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                  [ЈD̖]R595.2        [īIRa] A          [¾̖]1672-2353(2013)05-047-04 DOI:10.7619/jcmp.201305013

                  The Impact of Lead-x Oral Solution on Neurobehavioral Capabilities of Lead Exposing Children

                  1. Child Health Bureau, Children's Hospital of Capital Institute of Pediatrics, Beijing 100020China; 2. Pediatric Department, Beijing Anzhen Hospital of the Capital  University of Medical Sciences, Beijing 100029China

                  [Abstract] Objective: To investigate the impact of Lead-x oral solution on  blood lead levels and neurobehavioral capabilities in children exposed to low concentrations of lead.  Methods: Lead-x oral liquid was given to the children exposed to lead environment, and to observe its impact on children's blood lead levels and neurobehavioral capabilities.  Results: When the blood lead concentration was between 80g/L-100g/L, the visual reaction rate, the degree of focus and the memory span of reverse digital go down, and the error rate of auditory response increased simultaneously. When the blood lead concentration was between 100g/L-120g/L, Children showed further drop of the coordination of right hand and left foot cross movement, the memory span forward and reverse digital, and the motion stability. When the blood lead concentration was above 120g/L, 13 neurobehavioral capabilities are significantly affected. After taking Lead-x oral solution for 60 days, all children's blood lead level reduced to below 80g / L, and all neurobehavioral capabilities got varying degrees of improvement.  Conclusion: With increasing exposure of lead, aggravated damage on children's neurological function is discovered.  It is true that Lead-x oral solution could reduce blood lead level, and increase children's neurobehavioral capabilities.
                  [Key words] Lead-X oral solution; Children; Blood lead; Neurobehavioral capability


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                  īI
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                  [6] Gilbert SGWeissBA rationale for lowering the blood lead action level from 10 to 2g/dL [J]Neurotoxicology200627(5)693-
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                  [9] ѩ÷.ڷҺͯUѪY߹ܵӰ푣J.ˎWs־201218(2): 14-16.

                   

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